From the Archives

(1) A form of familial presenile dementia with spastic paralysis (including the pathological examination of a case). By C. Worster-Drought, J.G. Greenfield and W.H. McMenemey. Brain 1940: 63; 237–254. Plates VIII–XII. With (2) A form of familial presenile dementia with spastic paralysis. By C. Worster-Drought, J.G. Greenfield and W.H. McMenemey. Brain 1944: 67; 37–43. Plate 1. With (3) Subacute spongiform encephalopathy—a subacute form of encephalopathy attributable to vascular dysfunction (spongiform cerebral atrophy). By S. Nevin, W.H. McMenemey, S. Behrman and D.P Jones (From Maida Vale Hospital for Nervous Diseases and the Brook Hospital, London). Brain 1960: 83; 521–564. Plates LXXIX–LXXXIII. With (4) Creutzfeldt–Jakob disease. The neuropathology of a transmission experiment. By Elisabeth Beck, P.M. Daniel, W.B. Matthews, D.L. Stevens, M.P. Alpers, D.M. Asher, D.C. Gajdusek and C.J. Gibbs Jr (From the Department of Neuropathology, Institute of Psychiatry, The Maudsley Hospital, London, S.E.5, The Derbyshire Royal Infirmary, Derby, and National Institutes of Health, Bethesda, MD, USA). Brain 1969: 92; 699–716.With (5) Subacute spongiform encephalopathy (Creutzfeldt–Jakob disease). The nature and progression of spongiform change. By C.L. Masters and E.P Richardson Jr. (From the Charles S. Kubik Laboratory for Neuropathology, Massachusetts General Hospital, and the Departments of Neurology-Neuropathology, Harvard Medical School, Boston). Brain 1978: 101; 333–344. With (6) Creutzfeldt–Jakob disease virus isolations from the Gerstmann–Sträussler syndrome with an analysis of the various forms of amyloid plaque deposition in the virus-induced spongiform encephalopathies. By Colin L. Masters, D. Carleton Gajdusek and Clarence J. Gibbs Jr (From the Laboratory of Central Nervous System Studies, National Institute of Neurological and Communicative Disorders and Stroke, National Institutes of Health, Bethesda, MD, USA). Brain 1981: 104; 559–588.